Addressing the link between paraoxonase-1 gene variants and the incidence of early onset myocardial infarction

dc.AffiliationOctober University for modern sciences and Arts (MSA)
dc.contributor.authorF. Abdel Rahman, Mohamed
dc.contributor.authorM. Hashad, Ingy
dc.contributor.authorAbou-Aisha, Khaled
dc.contributor.authorM. Abdel-Maksoud, Sahar
dc.contributor.authorZ. Gad, Mohamed
dc.date.accessioned2020-01-23T09:18:44Z
dc.date.available2020-01-23T09:18:44Z
dc.date.issued2015
dc.descriptionMSA Google Scholaren_US
dc.description.abstractThe enzyme paraoxonase-1 (PON1) represents an endogenous defense mechanism against vascular oxidative stress, thereby contributing to the prevention of atherosclerosis. Several polymorphisms have been reported in the PON1 gene, including Q192R. PON1 phenotype is commonly expressed as the paraoxonase/arylesterase ratio (PON/ARE). The major aim of this study was
dc.titleAddressing the link between paraoxonase-1 gene variants and the incidence of early onset myocardial infarction
dc.typeArticle

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